Lactic Acid? Tied Up?
A Significant Advancement For High
Still No 1 Since
LACTAGON™ is a comprehensive supplement that
reduces lactic acid production, increasing energy &
muscle function for top performance and strong
LACTAGON™, powder (34 gm) and paste (1.5 oz tube in honey base, has been
used and proven on race tracks and in show arenas. LACTAGON™ is a
constant you can count on for your horses. It is a comprehensive
supplement, expressly formulate to provided essential nutrients for the
formation of acetyl coenzyme A in the equine athletes body. This is
important as it continues into the Citric Acid Cycle (also called the
Kreb’s Cycle). This cycle stores energy, released by the oxidation of
fats, proteins and carbohydrates in high energy phosphate bonds of ATP.
About 90% of the energy released occurs in this Citric Acid Cycle.
With this energy storage, then released during high performance exercise,
lactic acid production should be reduced and energy levels and muscle
function should be maintained for top competitive strong finishes.
From Owners And Trainers Who Are Using LACTAGON™
|"More staying power and
plenty of reserve for heading home."
||"My horses perform
truer to their form."
|"Brings out true
||"I use it while hauling
to reduce stress."
|"Fewer tying up
nervousness, better focus."
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LACTAGON™ Label including all ingredients!
The Severity of Metabolic Lactic Acidosis With Resultant Limitations Upon
Equine Exercise Physiology
Although it is recognized that the incidence of lactic acidosis in horses
always occurs during intense exercise and competition, there remains some
question pertaining to the detrimental effects within the scope of
decreasing the potential of a horse’s ability, and, to even a greater
degree, what are the manifestations of lactic acidosis and to what extent
is permanent damage incurred.
Lactic Acidosis is classified as an acute metabolic imbalance that is the
result of a deprivation of oxygen systemically. Aerobic oxidation of
metabolic chemical conversions (glycolysis) provides the release of
energy. And when an oxygen deficiency prevails termination of metabolic
activity occurs rapidly and the products of metabolism are deprived to the
body. Although an insufficient quantity of oxygen affects every chemical
conversion, at the metabolite, pyruvate, oxygen deficiency becomes a
crucial and pivoting element. Pyruvate is positioned as the final
metabolite, and at this juncture, in the presence of oxygen, the elements
of energy are made available; However, when stored oxygen is depleted,
then this metabolite, converts to lactic acid. The biosynthesis of lactic
acid is a chemical process where lactate dehydrogenase and Nicotinamide
Adenine Dinucleotide (NAD) participate in the exchange of hydrogen for
oxygen. In the biosynthesis of lactic acid, it is to be stored until
sufficient oxygen is available for reconversion to pyruvate.
The incidence of acute lactic acidosis is directly attributable to the
following equine disorders: Exercise related fatigue, muscle contraction,
pulmonary stress, diaphragm fatigue, fatigue related injuries, tendon
injuries, intracellular muscle damage, stress, and incurrence of numerous
drugs for treatment of the maladies and injuries as identified.
With respect towards the very serious physiological effects of acute
lactic acidosis, it is, therefore, highly desirable to bring about a means
of preventing or inhibiting the biosynthesis of lactic acid. To fully
understand the magnitude of this metabolic imbalance, lactic acidosis
should be evaluated as it affects organ and tissue function.
The pulmonary muscles, which encompass the lungs, are highly susceptible
to the condition of lactic acidosis. When muscle constriction occurs,
there is also a corresponding restrictive force placed upon the lungs.
During periods of intense exercise the lungs are subjected to
extraordinary stress due to this constraining force. Therefore, it is very
likely that an elevated level of lactic acidosis is responsible for the
occurrence of capillary and venule rupturing within the lungs and bleeding
from the lungs.
The effect of lactic acidosis upon muscle tissue is highly visible in the
form of a ”tyingup” syndrome. The contraction of muscle tissue is a result
of biochemical changes that are initiated by the formation of lactic acid.
An escalation of lactic acid within the tissues alters the utilization of
fuel and deprives muscle tissue of metabolic products essential for
sustaining muscle use.
Exercise Related Fatigue
In the presence of an aerobic phase of exercise, the formation and release
of energy elements is severely restricted. With oxygen, one glucose
molecule will form four CO and four HO t thirty eight ATP. With the
absence of oxygen to facilitate the metabolic conversions, one glucose
molecule will produce only eight ATP.
Lactic acid introduces changes within the cell that interfere with normal
physiological processes. These changes are known to gradually after
cellular function on a permanent basis. With a loss of cellular function
within the muscle tissue comes muscle atrophy and necrosis, as is
evidenced in myocardial infarction. This is very important in
understanding the extreme severity of lactic acidosis in that it is the
primary factor in decreasing muscle capability. In equine application,
this translates to a lessening of ability as muscles become more damaged
due to lactic acid.
It is recognized that a problem occurring in the transport of horses is
the development of stress. Thorough examinations of induced stress have
been performed in analyzing a relationship of stress to racing ability. It
is known that there is a definite correlation between transport
attributable stress and a diminishment of performance potential. It is
known that lactic acidosis emerges from physiological disruptions, such as
severe stress, and that the transported horse is likely to suffer.
As metabolic lactic acidosis is clearly responsible for the constriction
of muscle tissue, it is then logical that it is also a factor in tendon
injuries. Tendons are peripherally supported by muscles and when such
muscles contract then there is a significant increase in stress placed
upon the tendon. It is likely that the complications of mechanical stress
placed upon tendons may be substantially reduced through lowering the
occurrence of acute lactic acidosis.
It is well worth noting that the history of injuries occurring to horses
during racing reflects that most serious injuries do happen during a
period of exhaustion. As exhaustion is the culmination of the biochemical
changes due to lactic acid build-up, to circumvent lactic acidosis may
reduce instances of exhaustion and therefore diminish the probability that
a horse will suffer a career threatening injury.
The diaphragm muscles require an extraordinary amount of oxygen and to
satisfy these requirements this muscle group receives seven times the
normal blood supply to other muscle tissue. Under an anaerobic debt
incurred during intense exercise the diaphragm is also most sensitive to
lactic acid. Lactic acid build-up within the diaphragm results with not
only muscle constriction, thereby restricting air flow to the lungs, but
contributes towards a gradual and permanent loss of diaphragm function. It
is reasonable to consider that constriction of the diaphragm and some loss
of function may be contributing factors in pulmonary distress.
LACTAGON™ is a nutritional supplement and not a drug.
LACTAGON™ is a trademark of Wil-Bar Inc.
PO Box 461
McColl, South Carolina 29570 USA
910-276-3616 | fax-910-276-3616
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Toll Free - 800-831-1077
These products are not intended to
diagnose, treat, cure or prevent any disease.
*These statements have not been evaluated by the FDA.
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