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Introducing Our Brand New Product! - Lightning in a Tube! Click Here for Details!!
 

Lactagon

Lactic Acid?  Tied Up?  Nervous?  Quits?

A Significant Advancement For High Performance Horses

Still No 1 Since 1990!

Lactagon Horse Supplement

LACTAGON™ is a comprehensive supplement that reduces lactic acid production, increasing energy & muscle function for top performance and strong finishes!

LACTAGON™, powder (34 gm) and paste (1.5 oz tube in honey base, has been used and proven on race tracks and in show arenas.  LACTAGON™ is a constant you can count on for your horses. It is a comprehensive supplement, expressly formulate to provided essential nutrients for the formation of acetyl coenzyme A in the equine athletes body. This is important as it continues into the Citric Acid Cycle (also called the Kreb’s Cycle). This cycle stores energy, released by the oxidation of fats, proteins and carbohydrates in high energy phosphate bonds of ATP.  About 90% of the energy released occurs in this Citric Acid Cycle.  With this energy storage, then released during high performance exercise, lactic acid production should be reduced and energy levels and muscle function should be maintained for top competitive strong finishes.

Comments From Owners And Trainers Who Are Using LACTAGON™
"More staying power and plenty of reserve for heading home." "My horses perform truer to their form."
"Brings out true potential." "I use it while hauling to reduce stress."
"Fewer tying up problems." "Helps reduce nervousness, better focus."
"Relaxed energy." "Soothed irritability, especially restlessness."

Click Here to view the entire LACTAGON™ Label including all ingredients!
 


White Paper:
The Severity of Metabolic Lactic Acidosis With Resultant Limitations Upon Equine Exercise Physiology

Although it is recognized that the incidence of lactic acidosis in horses always occurs during intense exercise and competition, there remains some question pertaining to the detrimental effects within the scope of decreasing the potential of a horse’s ability, and, to even a greater degree, what are the manifestations of lactic acidosis and to what extent is permanent damage incurred.

Lactic Acidosis is classified as an acute metabolic imbalance that is the result of a deprivation of oxygen systemically. Aerobic oxidation of metabolic chemical conversions (glycolysis) provides the release of energy. And when an oxygen deficiency prevails termination of metabolic activity occurs rapidly and the products of metabolism are deprived to the body. Although an insufficient quantity of oxygen affects every chemical conversion, at the metabolite, pyruvate, oxygen deficiency becomes a crucial and pivoting element. Pyruvate is positioned as the final metabolite, and at this juncture, in the presence of oxygen, the elements of energy are made available; However, when stored oxygen is depleted, then this metabolite, converts to lactic acid. The biosynthesis of lactic acid is a chemical process where lactate dehydrogenase and Nicotinamide Adenine Dinucleotide (NAD) participate in the exchange of hydrogen for oxygen. In the biosynthesis of lactic acid, it is to be stored until sufficient oxygen is available for reconversion to pyruvate.

The incidence of acute lactic acidosis is directly attributable to the following equine disorders: Exercise related fatigue, muscle contraction, pulmonary stress, diaphragm fatigue, fatigue related injuries, tendon injuries, intracellular muscle damage, stress, and incurrence of numerous drugs for treatment of the maladies and injuries as identified.

With respect towards the very serious physiological effects of acute lactic acidosis, it is, therefore, highly desirable to bring about a means of preventing or inhibiting the biosynthesis of lactic acid. To fully understand the magnitude of this metabolic imbalance, lactic acidosis should be evaluated as it affects organ and tissue function.

Pulmonary Stress
The pulmonary muscles, which encompass the lungs, are highly susceptible to the condition of lactic acidosis. When muscle constriction occurs, there is also a corresponding restrictive force placed upon the lungs. During periods of intense exercise the lungs are subjected to extraordinary stress due to this constraining force. Therefore, it is very likely that an elevated level of lactic acidosis is responsible for the occurrence of capillary and venule rupturing within the lungs and bleeding from the lungs.

Muscle Contraction
The effect of lactic acidosis upon muscle tissue is highly visible in the form of a ”tyingup” syndrome. The contraction of muscle tissue is a result of biochemical changes that are initiated by the formation of lactic acid. An escalation of lactic acid within the tissues alters the utilization of fuel and deprives muscle tissue of metabolic products essential for sustaining muscle use.

Exercise Related Fatigue
In the presence of an aerobic phase of exercise, the formation and release of energy elements is severely restricted. With oxygen, one glucose molecule will form four CO and four HO t thirty eight ATP. With the absence of oxygen to facilitate the metabolic conversions, one glucose molecule will produce only eight ATP.

Muscle Damage
Lactic acid introduces changes within the cell that interfere with normal physiological processes. These changes are known to gradually after cellular function on a permanent basis. With a loss of cellular function within the muscle tissue comes muscle atrophy and necrosis, as is evidenced in myocardial infarction. This is very important in understanding the extreme severity of lactic acidosis in that it is the primary factor in decreasing muscle capability. In equine application, this translates to a lessening of ability as muscles become more damaged due to lactic acid.

Stress
It is recognized that a problem occurring in the transport of horses is the development of stress. Thorough examinations of induced stress have been performed in analyzing a relationship of stress to racing ability. It is known that there is a definite correlation between transport attributable stress and a diminishment of performance potential. It is known that lactic acidosis emerges from physiological disruptions, such as severe stress, and that the transported horse is likely to suffer.

Tendon Injuries
As metabolic lactic acidosis is clearly responsible for the constriction of muscle tissue, it is then logical that it is also a factor in tendon injuries. Tendons are peripherally supported by muscles and when such muscles contract then there is a significant increase in stress placed upon the tendon. It is likely that the complications of mechanical stress placed upon tendons may be substantially reduced through lowering the occurrence of acute lactic acidosis.

Injuries
It is well worth noting that the history of injuries occurring to horses during racing reflects that most serious injuries do happen during a period of exhaustion. As exhaustion is the culmination of the biochemical changes due to lactic acid build-up, to circumvent lactic acidosis may reduce instances of exhaustion and therefore diminish the probability that a horse will suffer a career threatening injury.

Air Restriction
The diaphragm muscles require an extraordinary amount of oxygen and to satisfy these requirements this muscle group receives seven times the normal blood supply to other muscle tissue. Under an anaerobic debt incurred during intense exercise the diaphragm is also most sensitive to lactic acid. Lactic acid build-up within the diaphragm results with not only muscle constriction, thereby restricting air flow to the lungs, but contributes towards a gradual and permanent loss of diaphragm function. It is reasonable to consider that constriction of the diaphragm and some loss of function may be contributing factors in pulmonary distress.

LACTAGON™ is a nutritional supplement and not a drug.
LACTAGON™ is a trademark of Wil-Bar Inc.

 



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McColl, South Carolina  29570  USA
910-276-3616 | fax-910-276-3616
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These products are not intended to diagnose, treat, cure or prevent any disease.
*These statements have not been evaluated by the FDA.
 


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